Dolores Corella, Oscar Coltell, George Mattingley, José V. Sorlí, Jose M. Ordovas. . (2017) Utilizing nutritional genomics to tailor diets for the prevention of cardiovascular disease: a guide for upcoming studies and implementations. 17:5, 495-513.
There are a variety of uncommon causes, but the diseasehas nearly vanished thanks to the elimination ofmumps,which crossed the placenta and infected the unborn child (Circulation 95: 133, 1997.)Especially in central Africa, there's a relatively common, similar lesion centered on the apex.
James M. Rippe, Theodore J. Angelopoulos. . 2017. The Role of Nutrition and Lifestyle in the Prevention and Treatment of Cardiovascular Disease. Nutrition in Lifestyle Medicine, 137-150.
John Cawley, Chad Meyerhoefer, Leah G. Gillingham, Penny Kris-Etherton, Peter J. H. Jones. . (2017) Estimates of the direct and indirect cost savings associated with heart disease that could be avoided through dietary change in the United States. 20:2, 182-192.
I'm not big on the term ISCHEMIC CARDIOMYOPATHY for the fibrosisof the heart that's sustained many little infarcts, since the disease is notprimarily the muscle.
Dagfinn Aune, NaNa Keum, Edward Giovannucci, Lars T. Fadnes, Paolo Boffetta, Darren C. Greenwood, Serena Tonstad, Lars J. Vatten, Elio Riboli, Teresa Norat. . (2016) Nut consumption and risk of cardiovascular disease, total cancer, all-cause and cause-specific mortality: a systematic review and dose-response meta-analysis of prospective studies. 14:1.
Chesney Richter, Ann Skulas-Ray, Penny Kris-Etherton. . 2017. The Role of Diet in the Prevention and Treatment of Cardiovascular Disease. Nutrition in the Prevention and Treatment of Disease, 595-623.
We've already seen endocardial fibroelastosis as a mimic, and some of the diseasesof skeletal muscle can also produce a restrictive cardiomyopathy (Heart & Lung 40: e123, 2011).
99:1390, 2006), and since the pathophysiology different fundamentally from othercauses of heart muscle failure, "the usual medications" tend to make matters worse.
People who've been on hemodialysis for a long time often have calcium chunksin the heart; these can be huge but seldom cause problems (Medicine 91: 165, 2012).
There are a variety of empirical treatments, the disease can hang around for a long time,and several percent of women still die of itACROMEGALY (apoptosis of myocytes for some reason): Circulation 99:426, 1999.POLYMYOSITIS-DERMATOMYOSITIS can produce a cardiomyopathy,accelerate atherosclerosis, and/or promote widespread small-artery disease,depending on the patient (Rheumatology 45 S4: iv18, 2006.)
However, because of the heterogeneity of expression, the variety of genes,and the difficulty of analysis, genetic screening isn't going to happenany time soon (JACC 50: 1813, 2007; update Circulation 123: 2701, 2011;Heart 97: 530, 2011).
Todd J. Anderson, Jean Grégoire, Glen J. Pearson, Arden R. Barry, Patrick Couture, Martin Dawes, Gordon A. Francis, Jacques Genest, Steven Grover, Milan Gupta, Robert A. Hegele, David C. Lau, Lawrence A. Leiter, Eva Lonn, G.B. John Mancini, Ruth McPherson, Daniel Ngui, Paul Poirier, John L. Sievenpiper, James A. Stone, George Thanassoulis, Richard Ward. . (2016) 2016 Canadian Cardiovascular Society Guidelines for the Management of Dyslipidemia for the Prevention of Cardiovascular Disease in the Adult. 32:11, 1263-1282.
It is part of the"hypereosinophilic syndrome", a mixed bag of myeloproliferative diseases,worm infestations, and "idiopathic" -- regardlessof cause (if it can be found), Loeffler's now treatablewith the anti-IL-5 antibody mepolizumab (NEJM 358: 1215, 2008).
Loeffler's eosinophilic disease of the heart usually features fibrosisof the endocardium with a lot of eosinophils (often a restrictivecardiomyopathy).